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Extra info for Annual Review of Immunology Volume 22 2004
Example text
These findings hint to cell-specific glycosylation of ICAM-3 enabling only binding of subsets of cells (K. van Gisbergen, unpublished results). The other self-glycoprotein, ICAM-2, plays a central role in DC migration and homing to secondary lymphoid tissues. Although DC-SIGN binds to both ICAM2 and ICAM-3 under static conditions, only the DC-SIGN-ICAM-2 interaction resists the shear stresses encountered under physiological flow conditions (33). Remarkably, DC-SIGN behaves as a DC-specific rolling receptor for ICAM-2 and is thus functionally similar to the selectins, which are well known for their regulation of leukocyte rolling upon carbohydrate-structure recognition (74).
J. Immunol. 6:288–92 Hardy RR, Hayakawa K, Haaijman J, Herzenberg LA. 1982. B-cell subpopulations identified by two-colour fluorescence analysis. Nature 297:589–91 Hardy RR, Hayakawa K, Parks DR, Herzenberg LA. 1983. Demonstration of B-cell maturation in X-linked immunodeficient mice by simultaneous three-colour immunofluorescence. Nature 306:270–72 Hardy RR, Hayakawa K, Parks DR, Herzenberg LA. 1984. Murine B cell differentiation lineages. J. Exp. Med. 159:1169– 88 Hayakawa K, Hardy RR, Parks DR, Herzenberg LA.
J. Exp. Med. 159:1169– 88 Hayakawa K, Hardy RR, Parks DR, Herzenberg LA. 1983. The “Ly-1 B” cell subpopulation in normal immunodefective, and autoimmune mice. J. Exp. Med. 157:202–18 Li YS, Hayakawa K, Hardy RR. 1993. The regulated expression of B lineage associated genes during B cell differentiation in bone marrow and fetal liver. J. Exp. Med. 178:951–60 Herzenberg LA, Kantor AB. 1993. B-cell lineages exist in the mouse. Immunol. Today 14:79–83; discussion 8–90 Wortis HH, Berland R. 2001. Cutting edge commentary: origins of B-1 cells.
